Pancreatic insufficiency is characterized by impaired digestion, malabsorption, nutrient/enzyme deficiencies, nausea and abdominal discomfort. Another indicator of pancreatic insufficiency is the intestinal overgrowth of Candida, bacteria, Mycoplasmas, yeast, protozoa, and worms (often liver flukes) in the small intestine and colon, adrenal/thyroid fatigue, etc. The most common causes of pancreatitis are gallstones and frequent and excessive consumption of alcohol (80% of cases), and less common causes are drugs or medication. The onset of pain in the abdomen region usually occurs within a day of binge drinking or eating a large meal (take digestive enzymes before eating). The chronic form of pancreatitis can be triggered by one acute attack that damages the pancreatic duct. The damaged duct causes the pancreas to become inflamed. Scar tissue develops (Sclero-Tox) and the pancreas is slowly destroyed.
There are many ways in which the blood sugar system can become imbalanced: xenobiotics, hormonal imbalances, emotional stress, poor diet, lack of exercise and rest to name but a few. Most of symptoms are produced due to an excess of insulin or cortisol in the blood. The imbalances can be hyperglycemia (too high), hypoglycemia (too low) or dysglycemia (too high AND too low). Insulin is a hormone that helps move blood sugar into cells where it’s used for energy.
The human body wants blood glucose (blood sugar) maintained in a very narrow range. It is the production of insulin and glucagon by the pancreas which ultimately determines if a patient has diabetes, hypoglycemia, or some other sugar problem. (Of course the liver and adrenals have a significant part to play in all this).
When it comes to the orchestration of all endocrine activity the proper release, production, utilization of insulin, for instance; there is a direct association with cortisol. Insulin maintains the upper parameters of blood sugar; Cortisol maintains the lower parameters and keeps it from going to low